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Diabetic foot

High YieldFoot & AnklethinKbox SBA

Pathophysiology

Diabetic foot disease usually results from the interaction of:

  • peripheral neuropathy with loss of protective sensation
  • peripheral arterial disease
  • repetitive pressure and deformity
  • impaired wound healing
  • infection

Motor neuropathy may produce clawing and altered pressure distribution. Autonomic neuropathy contributes to dry skin and fissuring.

Assessment

History

Ask about:

  • duration and control of diabetes
  • previous ulceration or amputation
  • neuropathic symptoms
  • claudication or rest pain
  • footwear
  • smoking
  • renal disease and other vascular risk factors

Examination

Assess:

  • ulcer site, size, depth and surrounding tissue
  • callus and deformity
  • warmth, swelling and erythema
  • pedal pulses and capillary refill
  • protective sensation with a 10 g monofilament
  • vibration and proprioception when required

Infection

Infection is a clinical diagnosis. Features include erythema, warmth, swelling, tenderness or purulent discharge.

Deep tissue obtained after debridement is preferable to a superficial swab when microbiological sampling is required.

Osteomyelitis

Suspect osteomyelitis with:

  • a deep or chronic ulcer
  • exposed bone or a positive probe-to-bone test
  • persistent inflammatory markers
  • suggestive radiographic change

MRI is useful when the diagnosis or extent is uncertain.

Vascular assessment

Peripheral arterial disease must be actively sought. ABI can be misleadingly high in diabetes because of arterial calcification; toe pressures or other vascular studies may be more useful when perfusion is uncertain.

Management

Ulcer care

  • pressure off-loading
  • debridement of devitalised tissue
  • appropriate dressings
  • optimisation of glycaemic control and nutrition
  • treatment of infection when present

Vascular disease

Refer for vascular assessment and revascularisation when perfusion is inadequate for healing.

Surgery

Surgery may be required for:

  • drainage or debridement of infection
  • osteomyelitis not controlled by conservative measures
  • correction of deformity causing recurrent ulceration
  • non-salvageable infection or ischaemia

Charcot neuroarthropathy

An acutely warm, swollen neuropathic foot with little pain should raise concern for Charcot neuroarthropathy. Early off-loading and immobilisation are essential.

References

  1. Orthobullets. Diabetic Foot Ulcers.
  2. Orthobullets. Diabetic Charcot Neuropathy.

Written/reviewed by Kishore Puthezhath

Professor of Orthopaedics and Consultant Paediatric Orthopaedic Surgeon

FRCS (Tr & Orth) revision resource

Reviewed: September 2026